Progression of and risk factors for dental erosion and wedge-shaped defects over a 6-year period.
Identifieur interne : 000524 ( Main/Exploration ); précédent : 000523; suivant : 000525Progression of and risk factors for dental erosion and wedge-shaped defects over a 6-year period.
Auteurs : A. Lussi [Suisse] ; M. SchaffnerSource :
- Caries research [ 0008-6568 ]
Descripteurs français
- KwdFr :
- Adulte (MeSH), Adulte d'âge moyen (MeSH), Concentration en ions d'hydrogène (MeSH), Facteurs de risque (MeSH), Facteurs temps (MeSH), Femelle (MeSH), Humains (MeSH), Modèles linéaires (MeSH), Mâle (MeSH), Population rurale (MeSH), Population urbaine (MeSH), Répartition aléatoire (MeSH), Salive (composition chimique), Salive (métabolisme), Suisse (MeSH), Érosion dentaire (classification), Érosion dentaire (diagnostic), Érosion dentaire (étiologie), Évolution de la maladie (MeSH).
- MESH :
- composition chimique : Salive, Érosion dentaire.
- diagnostic : Érosion dentaire.
- métabolisme : Salive.
- étiologie : Érosion dentaire.
- Adulte, Adulte d'âge moyen, Concentration en ions d'hydrogène, Facteurs de risque, Facteurs temps, Femelle, Humains, Modèles linéaires, Mâle, Population rurale, Population urbaine, Répartition aléatoire, Suisse, Évolution de la maladie.
English descriptors
- KwdEn :
- Adult (MeSH), Disease Progression (MeSH), Female (MeSH), Humans (MeSH), Hydrogen-Ion Concentration (MeSH), Linear Models (MeSH), Male (MeSH), Middle Aged (MeSH), Random Allocation (MeSH), Risk Factors (MeSH), Rural Population (MeSH), Saliva (chemistry), Saliva (metabolism), Switzerland (MeSH), Time Factors (MeSH), Tooth Erosion (classification), Tooth Erosion (diagnosis), Tooth Erosion (etiology), Urban Population (MeSH).
- MESH :
- chemistry : Saliva.
- classification : Tooth Erosion.
- diagnosis : Tooth Erosion.
- etiology : Tooth Erosion.
- metabolism : Saliva.
- Adult, Disease Progression, Female, Humans, Hydrogen-Ion Concentration, Linear Models, Male, Middle Aged, Random Allocation, Risk Factors, Rural Population, Switzerland, Time Factors, Urban Population.
Abstract
It has been shown that dental erosion is not a very rare condition. Careful observation and clinical experience led us to hypothesize a progression of these lesions with time despite dietary counselling and oral hygiene instruction. The purpose of this study was to determine the progression rate and risk factors of dental erosion and wedge-shaped defects over a 6-year period. In the year 1987, 204 randomly selected persons living in Bern from two age groups (26-30 and 46-50 years) were examined for frequency and severity of dental erosion and wedge-shaped defects. In the year 1993, 55 persons could be re-examined by the same dentist using the same indices. Further, detailed case histories of the patients were recorded and the flow rate and buffering capacity of the saliva were determined. The results indicated a distinct progression of erosion on facial and occlusal surfaces and wedge-shaped defects over the 6-year period. The increase in the defects was more marked in the older age group. Surprisingly, nutritional habits did not change despite thorough informative discussion about erosive foodstuff with every patient during the first session. Multiple linear regression analysis revealed consumption of nutritional acids and age as significant risk factors explaining 28% of the variability of the progression of erosions. The significant factors for the progression of wedge-shaped defects were identified to be frequency of tooth brushing and age, explaining 21% of the variability of the progression of wedge-shaped defects. In summary, there was a definite statistically significant progression of non-carious dental hard tissue defects in our sample. This progression was correlated with age (erosion and wedge-shaped defects), consumption of dietary acids (erosion) and frequency of toothbrushing (wedge-shaped defects).
DOI: 10.1159/000016587
PubMed: 10773637
Affiliations:
Links toward previous steps (curation, corpus...)
Le document en format XML
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Lussi</name><affiliation wicri:level="4"><nlm:affiliation>Department of Operative, Preventive and Paediatric Dentistry, University of Bern, School of Dental Medicine, Bern, Switzerland.</nlm:affiliation><country xml:lang="fr">Suisse</country><wicri:regionArea>Department of Operative, Preventive and Paediatric Dentistry, University of Bern, School of Dental Medicine, Bern</wicri:regionArea><placeName><settlement type="city">Berne</settlement><region type="région" nuts="3">Canton de Berne</region></placeName><orgName type="university">Université de Berne</orgName></affiliation></author><author><name sortKey="Schaffner, M" sort="Schaffner, M" uniqKey="Schaffner M" first="M" last="Schaffner">M. 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Careful observation and clinical experience led us to hypothesize a progression of these lesions with time despite dietary counselling and oral hygiene instruction. The purpose of this study was to determine the progression rate and risk factors of dental erosion and wedge-shaped defects over a 6-year period. In the year 1987, 204 randomly selected persons living in Bern from two age groups (26-30 and 46-50 years) were examined for frequency and severity of dental erosion and wedge-shaped defects. In the year 1993, 55 persons could be re-examined by the same dentist using the same indices. Further, detailed case histories of the patients were recorded and the flow rate and buffering capacity of the saliva were determined. The results indicated a distinct progression of erosion on facial and occlusal surfaces and wedge-shaped defects over the 6-year period. The increase in the defects was more marked in the older age group. Surprisingly, nutritional habits did not change despite thorough informative discussion about erosive foodstuff with every patient during the first session. Multiple linear regression analysis revealed consumption of nutritional acids and age as significant risk factors explaining 28% of the variability of the progression of erosions. The significant factors for the progression of wedge-shaped defects were identified to be frequency of tooth brushing and age, explaining 21% of the variability of the progression of wedge-shaped defects. In summary, there was a definite statistically significant progression of non-carious dental hard tissue defects in our sample. This progression was correlated with age (erosion and wedge-shaped defects), consumption of dietary acids (erosion) and frequency of toothbrushing (wedge-shaped defects).</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">10773637</PMID><DateCompleted><Year>2000</Year><Month>06</Month><Day>21</Day></DateCompleted><DateRevised><Year>2018</Year><Month>11</Month><Day>30</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0008-6568</ISSN><JournalIssue CitedMedium="Print"><Volume>34</Volume><Issue>2</Issue><PubDate><MedlineDate>2000 Mar-Apr</MedlineDate></PubDate></JournalIssue><Title>Caries research</Title><ISOAbbreviation>Caries Res</ISOAbbreviation></Journal><ArticleTitle>Progression of and risk factors for dental erosion and wedge-shaped defects over a 6-year period.</ArticleTitle><Pagination><MedlinePgn>182-7</MedlinePgn></Pagination><Abstract><AbstractText>It has been shown that dental erosion is not a very rare condition. Careful observation and clinical experience led us to hypothesize a progression of these lesions with time despite dietary counselling and oral hygiene instruction. The purpose of this study was to determine the progression rate and risk factors of dental erosion and wedge-shaped defects over a 6-year period. In the year 1987, 204 randomly selected persons living in Bern from two age groups (26-30 and 46-50 years) were examined for frequency and severity of dental erosion and wedge-shaped defects. In the year 1993, 55 persons could be re-examined by the same dentist using the same indices. Further, detailed case histories of the patients were recorded and the flow rate and buffering capacity of the saliva were determined. The results indicated a distinct progression of erosion on facial and occlusal surfaces and wedge-shaped defects over the 6-year period. The increase in the defects was more marked in the older age group. Surprisingly, nutritional habits did not change despite thorough informative discussion about erosive foodstuff with every patient during the first session. Multiple linear regression analysis revealed consumption of nutritional acids and age as significant risk factors explaining 28% of the variability of the progression of erosions. The significant factors for the progression of wedge-shaped defects were identified to be frequency of tooth brushing and age, explaining 21% of the variability of the progression of wedge-shaped defects. In summary, there was a definite statistically significant progression of non-carious dental hard tissue defects in our sample. This progression was correlated with age (erosion and wedge-shaped defects), consumption of dietary acids (erosion) and frequency of toothbrushing (wedge-shaped defects).</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Lussi</LastName><ForeName>A</ForeName><Initials>A</Initials><AffiliationInfo><Affiliation>Department of Operative, Preventive and Paediatric Dentistry, University of Bern, School of Dental Medicine, Bern, Switzerland.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Schaffner</LastName><ForeName>M</ForeName><Initials>M</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>Switzerland</Country><MedlineTA>Caries Res</MedlineTA><NlmUniqueID>0103374</NlmUniqueID><ISSNLinking>0008-6568</ISSNLinking></MedlineJournalInfo><CitationSubset>D</CitationSubset><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000328" MajorTopicYN="N">Adult</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D018450" MajorTopicYN="N">Disease Progression</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006863" MajorTopicYN="N">Hydrogen-Ion Concentration</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D016014" MajorTopicYN="N">Linear Models</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008875" MajorTopicYN="N">Middle Aged</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D011897" MajorTopicYN="N">Random Allocation</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D012307" MajorTopicYN="N">Risk Factors</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D012424" MajorTopicYN="N">Rural Population</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D012463" MajorTopicYN="N">Saliva</DescriptorName><QualifierName UI="Q000737" MajorTopicYN="N">chemistry</QualifierName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D013557" MajorTopicYN="N">Switzerland</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D013997" MajorTopicYN="N">Time Factors</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D014077" MajorTopicYN="N">Tooth Erosion</DescriptorName><QualifierName UI="Q000145" MajorTopicYN="N">classification</QualifierName><QualifierName UI="Q000175" MajorTopicYN="Y">diagnosis</QualifierName><QualifierName UI="Q000209" MajorTopicYN="N">etiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D014505" MajorTopicYN="N">Urban Population</DescriptorName></MeshHeading></MeshHeadingList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2000</Year><Month>4</Month><Day>25</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2000</Year><Month>6</Month><Day>24</Day><Hour>11</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2000</Year><Month>4</Month><Day>25</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">10773637</ArticleId><ArticleId IdType="pii">16587</ArticleId><ArticleId IdType="doi">10.1159/000016587</ArticleId></ArticleIdList></PubmedData></pubmed><affiliations><list><country><li>Suisse</li></country><region><li>Canton de Berne</li></region><settlement><li>Berne</li></settlement><orgName><li>Université de Berne</li></orgName></list><tree><noCountry><name sortKey="Schaffner, M" sort="Schaffner, M" uniqKey="Schaffner M" first="M" last="Schaffner">M. Schaffner</name></noCountry><country name="Suisse"><region name="Canton de Berne"><name sortKey="Lussi, A" sort="Lussi, A" uniqKey="Lussi A" first="A" last="Lussi">A. Lussi</name></region></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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